DiseaseID 25889

GM2神经节苷脂贮积症AB变异型

Gm2-Gangliosidosis, Ab Variant

MSH2017_2016_08_12:A progressive neurodegenerative disorder that begins with muscle weakness, then progresses to startle reaction, retardation and seizures. It is characterized by the accumulation of G(M2) GANGLIOSIDE in

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Disease: 1Formula: 20Herb: 2Symptom: 4Target: 19Links: 50
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Record Fields

Scalar fields from the final disease record.

Disease Id
25889
Core Entity Id
118457
Source Entity Count
1
Preferred Name
Gm2-Gangliosidosis, Ab Variant
Name Cn
GM2神经节苷脂贮积症AB变异型
Name Pinyin
Gm2 Shen Jing Jie Gan Zhi Zhu Ji Zheng Ab Bian Yi Xing
Name En
Gm2-Gangliosidosis, Ab Variant
Name Latin
Bilingual Status
complete
Disease Type
Umls Disease Type
Disgenet Type
Mesh Class
Do Class
Hpo Class
Mesh Class Name
Hpo Class Name
Do Class Name
Disease Definition
MSH2017_2016_08_12:A progressive neurodegenerative disorder that begins with muscle weakness, then progresses to startle reaction, retardation and seizures. It is characterized by the accumulation of G(M2) GANGLIOSIDE in neurons that is caused by a lack of G(M2) ACTIVATOR PROTEIN function. The AB variant designation refers to the increase of both HEXOSAMINIDASE A and HEXOSAMINIDASE B in tissues that lack of G(M2) activator protein.
Version
v1,v2
Suppressed
No

Names

Preferred names, aliases, and source labels retained in the final schema.

Name
Gm2-Gangliosidosis, Ab Variant
Role
preferred

Cross References

Trusted external identifiers retained for this final record.

Umls
C0268275
Sym Map
SMDE00823
Etcm Disease
Gm2-Gangliosidosis, Ab Variant
Itcmdb Generated
ITX-DISEASE-1EA215230BB7

Attributes

Merged source attributes and domain-specific metadata.

Version
v1,v2
Suppress
0
Page Title
Disease Gm2-Gangliosidosis, Ab Variant Details page
Basic Information
Disease Name
Gm2-Gangliosidosis, Ab Variant
Global Category
Genetic diseases;Metabolic diseases;Rare diseases
Anatomical Category
Bone diseases;Eye diseases;Liver diseases;Neuronal diseases
Disease Definition
MSH2017_2016_08_12:A progressive neurodegenerative disorder that begins with muscle weakness, then progresses to startle reaction, retardation and seizures. It is characterized by the accumulation of G(M2) GANGLIOSIDE in neurons that is caused by a lack of G(M2) ACTIVATOR PROTEIN function. The AB variant designation refers to the increase of both HEXOSAMINIDASE A and HEXOSAMINIDASE B in tissues that lack of G(M2) activator protein.