ReferenceID 972

Natural compound catechol induces DNA damage, apoptosis, and G1 cell cycle arrest in breast cancer cells

Phytother Res

Targeting cell cycle and inducing DNA damage by activating cell death pathways are considered as effective therapeutic strategy for combating breast cancer progression. Many of the naturally known small molecules target

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Reference Id
972
Evidence Id
17562
Core Evidence Id
17562
Source Reference Id
1938
Herb2 Reference Id
HBREF002735
Subject Paper Key
HBIN000735_33289235
Pubmed Id
33289235
Doi
10.1002/ptr.6970
Paper Title
Natural compound catechol induces DNA damage, apoptosis, and G1 cell cycle arrest in breast cancer cells
Paper Abstract
Targeting cell cycle and inducing DNA damage by activating cell death pathways are considered as effective therapeutic strategy for combating breast cancer progression. Many of the naturally known small molecules target these signaling pathways and are effective against resistant and/or aggressive types of breast cancers. Here, we investigated the effect of catechol, a naturally occurring plant compound, for its specificity and chemotherapeutic efficacies in breast cancer (MCF-7 and MDA-MB-231) cells. Catechol treatment showed concentration-dependent cytotoxicity and antiproliferative growth in both MCF-7 and MDA-MB-231 cells while sparing minimal effects on noncancerous (F-180 and HK2) cells. Catechol modulated differential DNA damage effects by activating ATM/ATR pathways and showed enhanced gamma-H2AX expression, as an indicator for DNA double-stranded breaks. MCF-7 cells showed G1 cell cycle arrest by regulating p21-mediated cyclin E/Cdk2 inhibition. Furthermore, activation of p53 triggered a caspase-mediated cell death mechanism by inhibiting regulatory proteins such as DNMT1, p-BRCA1, MCL-1, and PDCD6 with an increased Bax/Bcl-2 ratio. Overall, our results showed that catechol possesses favorable safety profile for noncancerous cells while specifically targeting multiple signaling cascades to inhibit proliferation in breast cancer cells.
Journal
Phytother Res
Publish Year
2021
Experiment Subject
Experiment Type
Cell Experiment
Phenotype Related
Breast Cancers; Breast Cancer
Paper Title Cn
Paper Title En
Natural compound catechol induces DNA damage, apoptosis, and G1 cell cycle arrest in breast cancer cells
Bilingual Status
semi_complete