ReferenceID 6204

Sesamin protects against DSS-induced colitis in mice by inhibiting NF-κB and MAPK signaling pathways

Food Funct

OBJECTIVE: To investigate the protective effects and mechanisms of sesamin (SES) on dextran sulfate sodium (DSS)-induced experimental colitis in mice. METHODS: SES (50, 100, and 200 mg kg-1) were orally administered to C

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Reference Id
6204
Evidence Id
22794
Core Evidence Id
22794
Source Reference Id
5680
Herb2 Reference Id
HBREF006477
Subject Paper Key
HBIN043798_33496702
Pubmed Id
33496702
Doi
10.1039/d0fo00950d
Paper Title
Sesamin protects against DSS-induced colitis in mice by inhibiting NF-κB and MAPK signaling pathways
Paper Abstract
OBJECTIVE: To investigate the protective effects and mechanisms of sesamin (SES) on dextran sulfate sodium (DSS)-induced experimental colitis in mice. METHODS: SES (50, 100, and 200 mg kg-1) were orally administered to C57BL/6 male mice after DSS instillation. The anti-inflammatory effect of SES on colonic damage was assessed by clinical, macroscopic, microscopic, and inflammatory signaling pathways. RESULTS AND CONCLUSIONS: It could be found that bodyweight and colon length of mice treated with DSS was significantly decreased while that were increased by SES treatment. SES treatment reduced the DAI values and improved the histopathology of the colon in the DSS-treated mice. SES also reduced TNF-alpha, IL-1beta and IL-6 production caused by DSS. We also measured the expression of the phosphorylation of p65, IkappaB, p38, ERK and JNK protein and found that SES can alleviate colon damage via the NF-kappaB and MAPK signaling pathways. The findings of this study suggested that SES had anti-inflammatory effects on intestinal inflammation and can be used as a new therapeutic candidate for inflammatory bowel disease.
Journal
Food Funct
Publish Year
2021
Experiment Subject
mouse
Experiment Type
Animal Experiment
Phenotype Related
Intestinal Inflammation; Colitis; Inflammatory Bowel Disease
Paper Title Cn
Paper Title En
Sesamin protects against DSS-induced colitis in mice by inhibiting NF-κB and MAPK signaling pathways
Bilingual Status
semi_complete