ReferenceID 6103

Disturbed glucose and pyruvate metabolism in glaucoma with neuroprotection by pyruvate or rapamycin

Proc Natl Acad Sci U S A

Intraocular pressure-sensitive retinal ganglion cell degeneration is a hallmark of glaucoma, the leading cause of irreversible blindness. Here, we used RNA-sequencing and metabolomics to examine early glaucoma in DBA/2J

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Reference Id
6103
Evidence Id
22693
Core Evidence Id
22693
Source Reference Id
5475
Herb2 Reference Id
HBREF006272
Subject Paper Key
HBIN041411_33318177
Pubmed Id
33318177
Doi
10.1073/pnas.2014213117
Paper Title
Disturbed glucose and pyruvate metabolism in glaucoma with neuroprotection by pyruvate or rapamycin
Paper Abstract
Intraocular pressure-sensitive retinal ganglion cell degeneration is a hallmark of glaucoma, the leading cause of irreversible blindness. Here, we used RNA-sequencing and metabolomics to examine early glaucoma in DBA/2J mice. We demonstrate gene expression changes that significantly impact pathways mediating the metabolism and transport of glucose and pyruvate. Subsequent metabolic studies characterized an intraocular pressure (IOP)-dependent decline in retinal pyruvate levels coupled to dysregulated glucose metabolism prior to detectable optic nerve degeneration. Remarkably, retinal glucose levels were elevated 50-fold, consistent with decreased glycolysis but possibly including glycogen mobilization and other metabolic changes. Oral supplementation of the glycolytic product pyruvate strongly protected from neurodegeneration in both rat and mouse models of glaucoma. Investigating further, we detected mTOR activation at the mechanistic nexus of neurodegeneration and metabolism. Rapamycin-induced inhibition of mTOR robustly prevented glaucomatous neurodegeneration, supporting a damaging role for IOP-induced mTOR activation in perturbing metabolism and promoting glaucoma. Together, these findings support the use of treatments that limit metabolic disturbances and provide bioenergetic support. Such treatments provide a readily translatable strategy that warrants investigation in clinical trials.
Journal
Proc Natl Acad Sci U S A
Publish Year
2020
Experiment Subject
mouse; rat
Experiment Type
Animal Experiment
Phenotype Related
Neurodegeneration; Glaucoma; Irreversible Blindness; Optic Nerve Degeneration; Intraocular Pressure-sensitive Retinal Ganglion Cell Degeneration; Glaucomatous Neurodegeneration; Metabolic Disturbances
Paper Title Cn
Paper Title En
Disturbed glucose and pyruvate metabolism in glaucoma with neuroprotection by pyruvate or rapamycin
Bilingual Status
semi_complete