ReferenceID 5440

Hinokiflavone Inhibits MDM2 Activity by Targeting the MDM2-MDMX RING Domain

Biomolecules

The proto-oncogene MDM2 is frequently amplified in many human cancers and its overexpression is clinically associated with a poor prognosis. The oncogenic activity of MDM2 is demonstrated by its negative regulation of tu

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Reference Id
5440
Evidence Id
22030
Core Evidence Id
22030
Source Reference Id
4130
Herb2 Reference Id
HBREF004927
Subject Paper Key
HBIN029395_35625571
Pubmed Id
35625571
Doi
10.3390/biom12050643
Paper Title
Hinokiflavone Inhibits MDM2 Activity by Targeting the MDM2-MDMX RING Domain
Paper Abstract
The proto-oncogene MDM2 is frequently amplified in many human cancers and its overexpression is clinically associated with a poor prognosis. The oncogenic activity of MDM2 is demonstrated by its negative regulation of tumor suppressor p53 and the substrate proteins involved in DNA repair, cell cycle control, and apoptosis pathways. Thus, inhibition of MDM2 activity has been pursued as an attractive direction for the development of anti-cancer therapeutics. Virtual screening was performed using the crystal structure of the MDM2-MDMX RING domain dimer against a natural product library and identified a biflavonoid Hinokiflavone as a promising candidate compound targeting MDM2. Hinokiflavone was shown to bind the MDM2-MDMX RING domain and inhibit MDM2-mediated ubiquitination in vitro. Hinokiflavone treatment resulted in the downregulation of MDM2 and MDMX and induction of apoptosis in various cancer cell lines. Hinokiflavone demonstrated p53-dependent and -independent tumor-suppressive activity. This report provides biochemical and cellular evidence demonstrating the anti-cancer effects of Hinokiflavone through targeting the MDM2-MDMX RING domain.
Journal
Biomolecules
Publish Year
2022
Experiment Subject
human; cancer cell lines
Experiment Type
Cell Experiment
Phenotype Related
Cancer; Cancers; Tumor
Paper Title Cn
Paper Title En
Hinokiflavone Inhibits MDM2 Activity by Targeting the MDM2-MDMX RING Domain
Bilingual Status
semi_complete