ReferenceID 5223

Effect of Fumonisin B1 on Proliferation and Apoptosis of Intestinal Porcine Epithelial Cells

Toxins (Basel)

Fumonisin B 1 (FB 1 ), which is a mycotoxin produced by Fusarium moniliforme and Fusarium rotarum , has a number of toxic effects in animals. Moldy feed containing FB 1 can damage the intestine. In this study, we used in

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Record Fields

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Reference Id
5223
Evidence Id
21813
Core Evidence Id
21813
Source Reference Id
3719
Herb2 Reference Id
HBREF004516
Subject Paper Key
HBIN026409_35878209
Pubmed Id
35878209
Doi
10.3390/toxins14070471
Paper Title
Effect of Fumonisin B1 on Proliferation and Apoptosis of Intestinal Porcine Epithelial Cells
Paper Abstract
Fumonisin B 1 (FB 1 ), which is a mycotoxin produced by Fusarium moniliforme and Fusarium rotarum , has a number of toxic effects in animals. Moldy feed containing FB 1 can damage the intestine. In this study, we used intestinal porcine epithelial cells (IPEC-J2) as an in vitro model to explore the effects of FB 1 on cell cycle and apoptosis. The results showed that IPEC-J2 cells treated with 10, 20, and 40 μg/mL FB 1 for 48 h experienced different degrees of damage manifested as decreases in cell number and viability, as well as cell shrinkage and floating. In addition, FB 1 reduced cell proliferation and the mRNA and protein expression of proliferating cell nuclear antigen (PCNA), cyclin-dependent kinase 2 (CDK2), CDK4, cyclinD1, and cyclinE1. FB 1 blocked the cell cycle in the G1 phase. FB 1 also induced mitochondrial pathway apoptosis, reduced mitochondrial membrane potential, and promoted mRNA and protein expression of Caspase3, Caspase9, and Bax. The findings suggest that FB 1 can induce IPEC-J2 cell damage, block the cell cycle, and promote cell apoptosis.
Journal
Toxins (Basel)
Publish Year
2022
Experiment Subject
ipec-j2 cells
Experiment Type
Cell Experiment
Phenotype Related
Paper Title Cn
Paper Title En
Effect of Fumonisin B1 on Proliferation and Apoptosis of Intestinal Porcine Epithelial Cells
Bilingual Status
semi_complete