ReferenceID 4743

Bilobalide inhibits inflammation and promotes the expression of Aβ degrading enzymes in astrocytes to rescue neuronal deficiency in AD models

Transl Psychiatry

The pathogenesis of Alzheimer's disease (AD) involves multiple cell types including endothelial cells, glia, and neurons. It suggests that therapy against single target in single cell type may not be sufficient to treat

Back to Browse

Relationship Network

Interactive first-hop connections across herbs, ingredients, formulas, targets, diseases, symptoms, syndromes, evidence, and monographs.

Click a node to open it in a new tab
Ingredient: 1Reference: 1Links: 1
Arranging relationship network...

Record Fields

Scalar fields from the final reference record.

Reference Id
4743
Evidence Id
21333
Core Evidence Id
21333
Source Reference Id
2725
Herb2 Reference Id
HBREF003522
Subject Paper Key
HBIN018512_34671017
Pubmed Id
34671017
Doi
10.1038/s41398-021-01594-2
Paper Title
Bilobalide inhibits inflammation and promotes the expression of Aβ degrading enzymes in astrocytes to rescue neuronal deficiency in AD models
Paper Abstract
The pathogenesis of Alzheimer's disease (AD) involves multiple cell types including endothelial cells, glia, and neurons. It suggests that therapy against single target in single cell type may not be sufficient to treat AD and therapies with protective effects in multiple cell types may be more effective. Here, we comprehensively investigated the effects of bilobalide on neuroinflammation and Abeta degrading enzymes in AD cell model and mouse model. We find that bilobalide inhibits Abeta-induced and STAT3-dependent expression of TNF-alpha, IL-1beta, and IL-6 in primary astrocyte culture. Bilobalide also induces robust expression of Abeta degrading enzymes like NEP, IDE, and MMP2 to facilitate astrocyte-mediated Abeta clearance. Moreover, bilobalide treatment of astrocyte rescues neuronal deficiency in co-cultured APP/PS1 neurons. Most importantly, bilobalide reduces amyloid and inflammation in AD mouse brain. Taken together, the protective effects of bilobalide in in vitro cultures were fully recapitulated in in vivo AD mouse model. Our study supports that bilobalide has therapeutic potential for AD treatment.
Journal
Transl Psychiatry
Publish Year
2021
Experiment Subject
mouse; co-cultured app/ps1 neurons; in vitro cultures
Experiment Type
Animal Experiment
Phenotype Related
Neuronal Deficiency; Alzheimer's Disease
Paper Title Cn
Paper Title En
Bilobalide inhibits inflammation and promotes the expression of Aβ degrading enzymes in astrocytes to rescue neuronal deficiency in AD models
Bilingual Status
semi_complete