ReferenceID 4334

Corticosterone inhibits GAS6 to govern hair follicle stem-cell quiescence

Nature

Chronic, sustained exposure to stressors can profoundly affect tissue homeostasis, although the mechanisms by which these changes occur are largely unknown. Here we report that the stress hormone corticosterone-which is

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Reference Id
4334
Evidence Id
20924
Core Evidence Id
20924
Source Reference Id
1947
Herb2 Reference Id
HBREF002744
Subject Paper Key
HBIN001977_33790465
Pubmed Id
33790465
Doi
10.1038/s41586-021-03417-2
Paper Title
Corticosterone inhibits GAS6 to govern hair follicle stem-cell quiescence
Paper Abstract
Chronic, sustained exposure to stressors can profoundly affect tissue homeostasis, although the mechanisms by which these changes occur are largely unknown. Here we report that the stress hormone corticosterone-which is derived from the adrenal gland and is the rodent equivalent of cortisol in humans-regulates hair follicle stem cell (HFSC) quiescence and hair growth in mice. In the absence of systemic corticosterone, HFSCs enter substantially more rounds of the regeneration cycle throughout life. Conversely, under chronic stress, increased levels of corticosterone prolong HFSC quiescence and maintain hair follicles in an extended resting phase. Mechanistically, corticosterone acts on the dermal papillae to suppress the expression of Gas6, a gene that encodes the secreted factor growth arrest specific 6. Restoring Gas6 expression overcomes the stress-induced inhibition of HFSC activation and hair growth. Our work identifies corticosterone as a systemic inhibitor of HFSC activity through its effect on the niche, and demonstrates that the removal of such inhibition drives HFSCs into frequent regeneration cycles, with no observable defects in the long-term.
Journal
Nature
Publish Year
2021
Experiment Subject
mouse
Experiment Type
Animal Experiment
Phenotype Related
Paper Title Cn
Paper Title En
Corticosterone inhibits GAS6 to govern hair follicle stem-cell quiescence
Bilingual Status
semi_complete