ReferenceID 3609

HIV-1 infection of human macrophages directly induces viperin which inhibits viral production

Blood

Macrophages are key target cells for HIV-1. HIV-1(BaL) induced a subset of interferon-stimulated genes in monocyte-derived macrophages (MDMs), which differed from that in monocyte-derived dendritic cells and CD4 T cells,

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Reference Id
3609
Evidence Id
20199
Core Evidence Id
20199
Source Reference Id
519
Herb2 Reference Id
HBREF000870
Subject Paper Key
HBIN026385_22677126
Pubmed Id
22677126
Doi
10.1182/blood-2012-01-407395
Paper Title
HIV-1 infection of human macrophages directly induces viperin which inhibits viral production
Paper Abstract
Macrophages are key target cells for HIV-1. HIV-1(BaL) induced a subset of interferon-stimulated genes in monocyte-derived macrophages (MDMs), which differed from that in monocyte-derived dendritic cells and CD4 T cells, without inducing any interferons. Inhibition of type I interferon induction was mediated by HIV-1 inhibition of interferon-regulated factor (IRF3) nuclear translocation. In MDMs, viperin was the most up-regulated interferon-stimulated genes, and it significantly inhibited HIV-1 production. HIV-1 infection disrupted lipid rafts via viperin induction and redistributed viperin to CD81 compartments, the site of HIV-1 egress by budding in MDMs. Exogenous farnesol, which enhances membrane protein prenylation, reversed viperin-mediated inhibition of HIV-1 production. Mutagenesis analysis in transfected cell lines showed that the internal S-adenosyl methionine domains of viperin were essential for its antiviral activity. Thus viperin may contribute to persistent noncytopathic HIV-1 infection of macrophages and possibly to biologic differences with HIV-1-infected T cells.
Journal
Blood
Publish Year
2012
Experiment Subject
macrophages (mdms)
Experiment Type
Cell Experiment
Phenotype Related
Paper Title Cn
Paper Title En
HIV-1 infection of human macrophages directly induces viperin which inhibits viral production
Bilingual Status
semi_complete