ReferenceID 3602

Hispolon suppresses metastasis via autophagic degradation of cathepsin S in cervical cancer cells

Cell Death Dis

Hispolon, a phenolic compound isolated from Phellinus igniarius, induces apoptosis and anti-tumor effects in cancers. However, the molecular mechanism involved in hispolon-mediated tumor-suppressing activities observed i

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Reference Id
3602
Evidence Id
20192
Core Evidence Id
20192
Source Reference Id
503
Herb2 Reference Id
HBREF000849
Subject Paper Key
HBIN029437_28981104
Pubmed Id
28981104
Doi
10.1038/cddis.2017.459
Paper Title
Hispolon suppresses metastasis via autophagic degradation of cathepsin S in cervical cancer cells
Paper Abstract
Hispolon, a phenolic compound isolated from Phellinus igniarius, induces apoptosis and anti-tumor effects in cancers. However, the molecular mechanism involved in hispolon-mediated tumor-suppressing activities observed in cervical cancer is poorly characterized. Here, we demonstrated that treatment with hispolon inhibited cell metastasis in two cervical cancer cell lines. In addition, the downregulation of the lysosomal protease Cathepsin S (CTSS) was critical for hispolon-mediated suppression of tumor cell metastasis in both in vitro and in vivo models. Moreover, hispolon induced autophagy, which increased LC3 conversion and acidic vesicular organelle formation. Mechanistically, hispolon inhibited the cell motility of cervical cells through the extracellular signal-regulated kinase (ERK) pathway, and blocking of the ERK pathway reversed autophagy-mediated cell motility and CTSS inhibition. Our results indicate that autophagy is essential for decreasing CTSS activity to inhibit tumor metastasis by hispolon treatment in cervical cancer; this finding provides a new perspective on molecular regulation.
Journal
Cell Death Dis
Publish Year
2017
Experiment Subject
cervical cancer cell lines
Experiment Type
Cell Experiment
Phenotype Related
Paper Title Cn
Paper Title En
Hispolon suppresses metastasis via autophagic degradation of cathepsin S in cervical cancer cells
Bilingual Status
semi_complete