ReferenceID 3596

Crystal structures of human transthyretin complexed with glabridin

J Med Chem

Transthyretin (TTR) is a plasma protein implicated in human amyloid diseases. Several small molecules that bind to the thyroxine-binding site of TTR have been shown to stabilize the TTR tetramer and to inhibit amyloid f

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Record Fields

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Reference Id
3596
Evidence Id
20186
Core Evidence Id
20186
Source Reference Id
490
Herb2 Reference Id
HBREF000833
Subject Paper Key
HBIN027849_24422526
Pubmed Id
24422526
Doi
10.1021/jm401832j
Paper Title
Crystal structures of human transthyretin complexed with glabridin
Paper Abstract
Transthyretin (TTR) is a plasma protein implicated in human amyloid diseases. Several small molecules that bind to the thyroxine-binding site of TTR have been shown to stabilize the TTR tetramer and to inhibit amyloid fibril formation of TTR. Herein, we demonstrated that glabridin (Glab), a prenylated isoflavan isolated from Glycyrrhiza glabra L., inhibited aggregation of TTR in a thioflavin assay. The TTR-Glab complex structure revealed a novel binding mode including a CH-π interaction with A108 and a hydrogen bond with K15. A structural comparison with the wild type-apo structure revealed that the CH-π interaction with A108 was strengthened by the induced-fit conformational change upon Glab binding. Furthermore, the binding of Glab induced a rotation of the T119 side chain, and the inclusion of a water molecule, leading to stabilization of the dimer-dimer interface. These results demonstrate that Glab is a novel inhibitor of TTR fibrillization and suggest the molecular mechanism by which Glab binding stabilizes the tetramer.
Journal
J Med Chem
Publish Year
2014
Experiment Subject
Experiment Type
Others
Phenotype Related
Human Amyloid Diseases
Paper Title Cn
Paper Title En
Crystal structures of human transthyretin complexed with glabridin
Bilingual Status
semi_complete