ReferenceID 2537

Orientin reduces the inhibitory effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on adipogenic differentiation and insulin signaling pathway in murine 3T3-L1 adipocytes

Chem Biol Interact

2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) accumulates in human body, probably influencing adipocyte differentiation and causing various toxic effects, including wasting syndrome. Recently, orientin, a phenolic compound

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Reference Id
2537
Evidence Id
19127
Core Evidence Id
19127
Source Reference Id
5083
Herb2 Reference Id
HBREF005880
Subject Paper Key
HBIN038267_32044341
Pubmed Id
32044341
Doi
10.1016/j.cbi.2020.108978
Paper Title
Orientin reduces the inhibitory effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on adipogenic differentiation and insulin signaling pathway in murine 3T3-L1 adipocytes
Paper Abstract
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) accumulates in human body, probably influencing adipocyte differentiation and causing various toxic effects, including wasting syndrome. Recently, orientin, a phenolic compound abundant in natural health products, has been shown to have antioxidant properties. We investigated the protective effects of orientin against TCDD-induced adipocyte dysfunction and its underlying mechanisms. In this study, orientin suppressed TCDD-induced loss of lipid accumulation. Orientin inhibited TCDD-driven decreases in the levels of peroxisome proliferator-activated receptor gamma and adiponectin. Orientin also reduced TCDD-induced prostaglandin E2, and cytosolic phospholipase A2alpha levels, and increased TCDD-inhibited peroxisome proliferator-activated receptor gamma coactivator 1-alpha levels in 3T3-L1 adipocytes. TCDD reduced the levels of insulin receptor substrate 1 and glucose transporter 4, and decreased insulin-stimulated glucose uptake activity; however, orientin diminished these TCDD-induced effects. These results suggest that orientin may have beneficial effects on the prevention of TCDD-induced wasting syndrome and type II diabetes mellitus accompanied by insulin resistance.
Journal
Chem Biol Interact
Publish Year
2020
Experiment Subject
human; 3t3-l1 adipocytes
Experiment Type
Cell Experiment
Phenotype Related
Adipocyte Dysfunction; Tcdd-induced Wasting Syndrome; Wasting Syndrome; Type Ii Diabetes Mellitus
Paper Title Cn
Paper Title En
Orientin reduces the inhibitory effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on adipogenic differentiation and insulin signaling pathway in murine 3T3-L1 adipocytes
Bilingual Status
semi_complete