ReferenceID 2283

Mangiferin alleviates hyperglycemia-induced endothelial impairment via Nrf2 signaling pathway

Eur J Pharmacol

The regulation of angiogenesis by nuclear factor erythroid 2-related factor 2 (Nrf2), a master redox transcription factor, is well established. In the present study, we aimed to activate Nrf2 by mangiferin (MG) and inves

Back to Browse

Relationship Network

Interactive first-hop connections across herbs, ingredients, formulas, targets, diseases, symptoms, syndromes, evidence, and monographs.

Click a node to open it in a new tab
Ingredient: 1Reference: 1Links: 1
Arranging relationship network...

Record Fields

Scalar fields from the final reference record.

Reference Id
2283
Evidence Id
18873
Core Evidence Id
18873
Source Reference Id
4593
Herb2 Reference Id
HBREF005390
Subject Paper Key
HBIN034394_36332683
Pubmed Id
36332683
Doi
10.1016/j.ejphar.2022.175359
Paper Title
Mangiferin alleviates hyperglycemia-induced endothelial impairment via Nrf2 signaling pathway
Paper Abstract
The regulation of angiogenesis by nuclear factor erythroid 2-related factor 2 (Nrf2), a master redox transcription factor, is well established. In the present study, we aimed to activate Nrf2 by mangiferin (MG) and investigate its potential to regulate angiogenesis under a hyperglycemic (HG) environment in human endothelial cells. The mRNA expression of Nrf2 and its downstream targets HO-1, SOD-1, and CAT were observed to be decreased at 72 h of HG (33.3 mM glucose) exposure, and was ameliorated in MG (24 h) pretreated endothelial cells. ROS generation was assessed using an DCFDA assay, where we found the ROS generated at HG exposure was quenched by MG in a dose-dependent manner. The angiogenic markers HIF-1α and VEGF were also decreased in HG-induced endothelial cells, which significantly increased in the cells pretreated with MG. In addition, we have observed substantially more closed tube network formation in MG pretreated cells, which was low in HG-induced endothelial cells. The cell migration potential was monitored using a scratch assay, where the cells activated by MG showed a more significant movement than those under HG stress. Furthermore, to understand the role of Nrf2 in regulating angiogenesis, we knocked out the Nrf2 using CRISPR/Cas9 in endothelial cells. The wild-type endothelial cells exposed to MG alone showed a cytoprotective effect under a hyperglycemic environment. Our findings collectively demonstrated that MG helps regulate impaired angiogenesis under a hyperglycemic environment through Nrf2 signaling.
Journal
Eur J Pharmacol
Publish Year
2022
Experiment Subject
human; hg-induced endothelial cells; mg pretreated cells; wild-type endothelial cells
Experiment Type
Cell Experiment
Phenotype Related
Paper Title Cn
Paper Title En
Mangiferin alleviates hyperglycemia-induced endothelial impairment via Nrf2 signaling pathway
Bilingual Status
semi_complete