ReferenceID 2129

Verbascoside and isoverbascoside ameliorate transforming growth factor β1-induced collagen expression by lung fibroblasts through Smad/non-Smad signaling pathways

Life Sci

Aims: Pulmonary fibrosis (PF) is a chronic, irreversible, and debilitating lung disease that typically leads to respiratory failure, and is a major cause of morbidity and mortality. Few drugs are effective for the treatm

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Reference Id
2129
Evidence Id
18719
Core Evidence Id
18719
Source Reference Id
4292
Herb2 Reference Id
HBREF005089
Subject Paper Key
HBIN031342_36100079
Pubmed Id
36100079
Doi
10.1016/j.lfs.2022.120950
Paper Title
Verbascoside and isoverbascoside ameliorate transforming growth factor β1-induced collagen expression by lung fibroblasts through Smad/non-Smad signaling pathways
Paper Abstract
Aims: Pulmonary fibrosis (PF) is a chronic, irreversible, and debilitating lung disease that typically leads to respiratory failure, and is a major cause of morbidity and mortality. Few drugs are effective for the treatment of patients with PF or for reducing the rate of disease progression. Main methods: Transforming growth factor-β1 (TGF-β1) is a profibrotic cytokine that signals through Smad and non-Smad pathways. Verbascoside (VB) and isoverbascoside (isoVB) exhibit anti-oxidative and anti-inflammatory activities, however, their anti-fibrotic effects remain unclear. This study evaluated the effects of VB and isoVB on TGF-β1-stimulated murine lung fibroblasts (MLg 2908) and also human lung fibroblasts (confirmed by immunostaining). Key findings: Neither VB nor isoVB had a cytotoxic effect on MLg 2908 fibroblasts. Both compounds (10 μM) reduced intracellular reactive oxygen species and markedly attenuated collagen I expression in TGF-β1 (5 ng/ml)-induced MLg 2908 cells compared to TGF-β1 alone. Both compounds suppressed the TGF-β1-induced phosphorylation of Smad2/3 and ERK/p38 mitogen-activated protein kinases (MAPKs). VB and isoVB, but not pirfenidone and nintedanib, inhibited TGF-β1-induced pSmad2/3, ERK/p38 MAPK, and collagen I expression. VB and isoVB also decreased collagen I deposition in TGF-β1-induced MLg 2908 cells. Only isoVB significantly suppressed collagen I deposition in TGF-β1-induced human pulmonary cells. Our results indicated that VB and isoVB may exert antifibrotic effects by inhibiting TGF-β1-induced collagen I expression via inhibition of oxidative stress and downregulation of the Smad/non-Smad pathway. Significance: The present findings suggest that VB or isoVB may be used as a supplement to alleviate PF.
Journal
Life Sci
Publish Year
2022
Experiment Subject
mouse; human; patient; mlg 2908; mlg 2908 cells; mlg 2908 fibroblasts; tgf-β1-induced human pulmonary cells; tgf-β1-induced mlg 2908 cells; tgf-β1-stimulated murine lung fibroblasts
Experiment Type
Cell Experiment
Phenotype Related
Pulmonary Fibrosis; Chronic, Irreversible, And Debilitating Lung Disease; Respiratory Failure
Paper Title Cn
Paper Title En
Verbascoside and isoverbascoside ameliorate transforming growth factor β1-induced collagen expression by lung fibroblasts through Smad/non-Smad signaling pathways
Bilingual Status
semi_complete