DiseaseID 28360

东方马脑脊髓炎

Eastern Equine Encephalomyelitis

MSH2017_2016_08_12:A form of arboviral encephalitis (primarily affecting equines) endemic to eastern regions of North America. The causative organism (ENCEPHALOMYELITIS VIRUS, EASTERN EQUINE) may be transmitted to humans

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Disease: 1Symptom: 4Target: 9Links: 13
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Record Fields

Scalar fields from the final disease record.

Disease Id
28360
Core Entity Id
120928
Source Entity Count
1
Preferred Name
Eastern Equine Encephalomyelitis
Name Cn
东方马脑脊髓炎
Name Pinyin
Dong Fang Ma Nao Ji Sui Yan
Name En
Eastern Equine Encephalomyelitis
Name Latin
Bilingual Status
complete
Disease Type
Umls Disease Type
Disgenet Type
Mesh Class
Do Class
Hpo Class
Mesh Class Name
Hpo Class Name
Do Class Name
Disease Definition
MSH2017_2016_08_12:A form of arboviral encephalitis (primarily affecting equines) endemic to eastern regions of North America. The causative organism (ENCEPHALOMYELITIS VIRUS, EASTERN EQUINE) may be transmitted to humans via the bite of AEDES mosquitoes. Clinical manifestations include the acute onset of fever, HEADACHE, altered mentation, and SEIZURES followed by coma. The condition is fatal in up to 50% of cases. Recovery may be marked by residual neurologic deficits and EPILEPSY. (From Joynt, Clinical Neurology, 1996, Ch26, pp9-10)
Version
v2
Suppressed
No

Names

Preferred names, aliases, and source labels retained in the final schema.

Name
Eastern Equine Encephalomyelitis
Role
preferred
Source
SymMap_v2
Preferred
Yes

Cross References

Trusted external identifiers retained for this final record.

Umls
C0153065
Sym Map
SMDE08122

Attributes

Merged source attributes and domain-specific metadata.

Version
v2
Suppress
0
Disease Definition
MSH2017_2016_08_12:A form of arboviral encephalitis (primarily affecting equines) endemic to eastern regions of North America. The causative organism (ENCEPHALOMYELITIS VIRUS, EASTERN EQUINE) may be transmitted to humans via the bite of AEDES mosquitoes. Clinical manifestations include the acute onset of fever, HEADACHE, altered mentation, and SEIZURES followed by coma. The condition is fatal in up to 50% of cases. Recovery may be marked by residual neurologic deficits and EPILEPSY. (From Joynt, Clinical Neurology, 1996, Ch26, pp9-10)